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the transient increase of tight junction permeability induced by bryostatin 1 correlates with rapid downregulation of protein kinase C-alpha
stresses
Makarov.
временное увеличение проницаемости плотных контактов, индуцированное бриостатином 1, коррелировано с быстрым снижением регуляции протеинкиназы c-альфа
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